Phone: (902) 494-3944
Fax: (902) 494-6585
- Vision impairment
- Cellular mechanisms
PhD (McMaster University)
PDF (Harvard Medical School)
Professor Duffy’s research aims to understand the neural events that permit sensory experience to shape brain development and function. His lab investigates cellular mechanisms that promote recovery from a vision impairment (amblyopia) caused by abnormal visual experience early in postnatal life.
USA Patent: PCT/US2016/046218
•Aronitz EM, Kamermans BA, Duffy KR (2021) Development of parvalbumin neurons and perineuronal nets in the visual cortex of normal and dark-exposed cats. Journal of Comparative Neurology, 529, 2827-2841.
•DiCostanzo NR, Crowder NA, Kamermans BA, Duffy KR (2020) Retinal and optic nerve integrity following monocular inactivation for the treatment of amblyopia. Frontiers in Systems Neuroscience 14:32.
•Lingley AJ, Mitchell DE, Crowder NA, Duffy KR (2019) Modification of peak plasticity induced by brief dark exposure. Neural Plasticity 3198285.
•Mitchell DE, Crowder NA, Duffy KR (2019) The critical period for darkness-induced recovery of the vision of the amblyopic eye following early monocular deprivation. Journal of vision 19: 25.
•Mitchell DE, Aronitz E, Bobbie-Ansah P, Crowder NA, Duffy KR (2019) Fast recovery of the amblyopic eye acuity of kittens following brief exposure to total darkness depends on the fellow eye. Neural Plasticity 7624837.
•Lingley AJ, Bowdridge JC, Farivar R, Duffy KR (2018) Mapping of neuron soma size as an effective approach to delineate differences between neural populations. Journal of Neuroscience Methods 304, 126-135.
•Duffy KR, Fong, MF, Mitchell DE, Bear MF (2018) Recovery from the anatomical effects of long-term monocular deprivation in cat lateral geniculate nucleus. Journal of Comparative Neurology 526, 310-323.
•Fong MF, Mitchell DE, Duffy KR, Bear MF (2016) Retinal inactivation promotes recovery from amblyopia. Proceedings of the National Academy of Sciences 213, 14139-14144.
•Duffy KR, Lingley AJ, Holman KD, Mitchell DE (2016) Susceptibility to monocular deprivation following immersion in darkness either late into or beyond the critical period. Journal of Comparative Neurology, 524, 2643-2653.